1 :

Chronic obstructive pulmonary disease (COPD) - diagnosed 10 years ago, Hypertension Hyperlipidemia, Former smoker: 40 pack-years, quit 2 years ago

Current Medications

Tiotropium inhaler once daily

Albuterol MDI as needed

Lisinopril 10 mg daily

Atorvastatin 20 mg nightly

Vitals: 100.6 *F, 112 bpm, 28/min labored, 152/88 mmHg, 86% on room air. Client is ALOx3. Client has diminished breath sounds with expiratory wheezing and accessory muscle use, prolonged expiratory phase. Productive cough with thick, green sputum. Heart rate 112bpm, regular rhythm. Bowel sounds present in all four quadrants. Voiding concentrated yellow urine with no complaints of pain. Skin clean, dry, and intact. Clubbing of fingernails present. 20G peripheral IV inserted into the right antecubital.

ABGsResultsReference
pH7.317.35-7.45
PaCO25835-45 mmHg
HCO33022-26 mEq/L
PaO25575-100 mmHg
CBCResultsReference
White blood cells14.54000-11000/mcL
Red blood cells5.8 million/mcL4.7-6.1 million/mcL
Hemoglobin13.1 g/dL13.2-16.6 g/dL
Hematocrit38%38.3-48.6%
Platelets152150-400 K/uL

Which clinical findings are most consistent with an acute COPD exacerbation? (Select all that apply)


Answer Options

Hint : 🌹 COPD worsens expiration and oxygenation

Correct Answers:

  • Prolonged expiratory phase with wheezing
  • Productive cough with yellow-green sputum
  • Sp02 86% on room air


Rationale :

✅ Prolonged expiratory phase with wheezing

🌹 COPD exacerbations cause worsening airflow obstruction, making it particularly difficult for the client to exhale effectively.

🌹 A prolonged expiratory phase is characteristic of obstructive pulmonary disease because narrowed airways slow the movement of air out of the lungs.

🌹 Expiratory wheezing results from air moving through narrowed or obstructed lower airways.

🌹 Increased bronchospasm and airway inflammation during an exacerbation can make wheezing more pronounced.

🌹 The client's diminished breath sounds further indicate decreased air movement associated with significant airflow obstruction.

🌹 The ABG showing PaCO₂ 58 mmHg supports impaired ventilation and carbon dioxide retention.

🌹 The client's labored respirations and accessory muscle use further indicate increased work of breathing during the acute episode.

🌹 Therefore, a prolonged expiratory phase with wheezing is strongly consistent with an acute COPD exacerbation.

✅ Productive cough with yellow-green sputum

🌹 An acute COPD exacerbation commonly presents with an increase in cough and sputum production.

🌹 A change in sputum characteristics, particularly development of purulent yellow-green sputum, can suggest an infectious trigger.

🌹 Respiratory infections are common precipitating factors for acute COPD exacerbations.

🌹 The client's low-grade fever of 100.6°F further supports the possibility of an acute infectious process.

🌹 The elevated WBC count of 14.5 also indicates an inflammatory or infectious response.

🌹 Increased airway secretions can worsen airflow obstruction and contribute to difficulty breathing.

🌹 The absence of focal consolidation on the chest x-ray makes focal pneumonia less evident, while the overall presentation remains consistent with COPD exacerbation.

🌹 Therefore, the client's productive cough with yellow-green sputum is an important finding associated with the acute exacerbation.

✅ SpO₂ 86% on room air

🌹 An SpO₂ of 86% on room air represents significant hypoxemia and is consistent with worsening gas exchange during an acute COPD exacerbation.

🌹 The client's ABG confirms hypoxemia with a PaO₂ of 55 mmHg.

🌹 Increased airway obstruction and ventilation-perfusion mismatch can significantly reduce oxygenation during an exacerbation.

🌹 The client's tachypnea of 28/min represents a compensatory response to respiratory compromise.

🌹 Accessory muscle use further demonstrates increased respiratory effort and difficulty maintaining adequate ventilation.

🌹 The ABG values of pH 7.31, PaCO₂ 58 mmHg, and HCO₃⁻ 30 mEq/L indicate respiratory acidosis with metabolic compensation, compatible with chronic CO₂ retention with acute worsening.

🌹 Significant hypoxemia requires prompt assessment and appropriately controlled supplemental oxygen therapy in a client with COPD.

🌹 Therefore, SpO₂ 86% on room air is a major clinical finding consistent with an acute COPD exacerbation.


Distractor Analysis :

Rationale for Incorrect Options to be Available Soon

2 :

Chronic obstructive pulmonary disease (COPD) - diagnosed 10 years ago, Hypertension Hyperlipidemia, Former smoker: 40 pack-years, quit 2 years ago

Current Medications

Tiotropium inhaler once daily

Albuterol MDI as needed

Lisinopril 10 mg daily

Atorvastatin 20 mg nightly

Vitals: 100.6 *F, 112 bpm, 28/min labored, 152/88 mmHg, 86% on room air. Client is ALOx3. Client has diminished breath sounds with expiratory wheezing and accessory muscle use, prolonged expiratory phase. Productive cough with thick, green sputum. Heart rate 112bpm, regular rhythm. Bowel sounds present in all four quadrants. Voiding concentrated yellow urine with no complaints of pain. Skin clean, dry, and intact. Clubbing of fingernails present. 20G peripheral IV inserted into the right antecubital.

ABGsResultsReference
pH7.317.35-7.45
PaCO25835-45 mmHg
HCO33022-26 mEq/L
PaO25575-100 mmHg
CBCResultsReference
White blood cells14.54000-11000/mcL
Red blood cells5.8 million/mcL4.7-6.1 million/mcL
Hemoglobin13.1 g/dL13.2-16.6 g/dL
Hematocrit38%38.3-48.6%
Platelets152150-400 K/uL

Which acid-base imbalance is reflected in the client's arterial blood gases (ABG)?


Answer Options

Hint : ✍️ Low pH + high CO₂ = respiratory acidosis

Correct Answers:

  • Respiratory alkalosis, uncompensated


Rationale :

✍️ The client's pH is 7.31, which is below the normal range of 7.35–7.45, indicating acidosis.

✍️ The PaCO₂ is 58 mmHg, which is above the normal range of 35–45 mmHg, indicating retention of carbon dioxide and identifying a respiratory cause of the acidosis.

✍️ Clients with COPD can retain carbon dioxide because airflow obstruction and impaired alveolar ventilation reduce effective elimination of CO₂.

✍️ The client's HCO₃⁻ is elevated at 30 mEq/L, compared with the normal range of 22–26 mEq/L, showing that the kidneys are retaining bicarbonate to compensate for the respiratory acidosis.

✍️ The compensation is only partial because the pH remains abnormal at 7.31 rather than returning to the normal range.

✍️ The client's PaO₂ of 55 mmHg and SpO₂ of 86% also demonstrate significant hypoxemia associated with impaired pulmonary gas exchange.

✍️ The acute worsening of COPD, including wheezing, prolonged expiration, tachypnea, and increased work of breathing, explains the worsening CO₂ retention.

✍️ Therefore, the combination of low pH + high PaCO₂ + high HCO₃⁻ identifies partially compensated respiratory acidosis.


Distractor Analysis :

Rationale for Incorrect Options to be Available Soon

3 :

Chronic obstructive pulmonary disease (COPD) - diagnosed 10 years ago, Hypertension Hyperlipidemia, Former smoker: 40 pack-years, quit 2 years ago

Current Medications

Tiotropium inhaler once daily

Albuterol MDI as needed

Lisinopril 10 mg daily

Atorvastatin 20 mg nightly

Vitals: 100.6 *F, 112 bpm, 28/min labored, 152/88 mmHg, 86% on room air. Client is ALOx3. Client has diminished breath sounds with expiratory wheezing and accessory muscle use, prolonged expiratory phase. Productive cough with thick, green sputum. Heart rate 112bpm, regular rhythm. Bowel sounds present in all four quadrants. Voiding concentrated yellow urine with no complaints of pain. Skin clean, dry, and intact. Clubbing of fingernails present. 20G peripheral IV inserted into the right antecubital.

ABGsResultsReference
pH7.317.35-7.45
PaCO25835-45 mmHg
HCO33022-26 mEq/L
PaO25575-100 mmHg
CBCResultsReference
White blood cells14.54000-11000/mcL
Red blood cells5.8 million/mcL4.7-6.1 million/mcL
Hemoglobin13.1 g/dL13.2-16.6 g/dL
Hematocrit38%38.3-48.6%
Platelets152150-400 K/uL

Which problem should the nurse prioritize for this client?


Answer Options

Hint : 👉 ABCs—breathing comes before infection

Correct Answers:

  • Ineffective breathing


Rationale :

👉 The client has severe respiratory compromise, including a respiratory rate of 28/min with labored breathing, accessory muscle use, wheezing, and prolonged expiration.

👉 The client's SpO₂ of 86% on room air demonstrates significant hypoxemia and indicates inadequate oxygenation.

👉 The PaO₂ of 55 mmHg further confirms impaired pulmonary gas exchange and insufficient arterial oxygenation.

👉 The PaCO₂ of 58 mmHg indicates carbon dioxide retention caused by inadequate alveolar ventilation associated with the COPD exacerbation.

👉 The client's pH of 7.31 demonstrates respiratory acidosis, showing that impaired ventilation is already producing a significant physiologic consequence.

👉 Using the ABC priority framework, breathing problems take priority over fluid balance, infection, and other concerns when respiratory compromise is present.

👉 The client's wheezing, diminished breath sounds, and accessory muscle use indicate airway obstruction and increased work of breathing requiring prompt intervention.

👉 Therefore, ineffective breathing is the priority problem because failure to improve ventilation and oxygenation can rapidly become life-threatening.


Distractor Analysis :

Rationale for Incorrect Options to be Available Soon

4 :

Chronic obstructive pulmonary disease (COPD) - diagnosed 10 years ago, Hypertension Hyperlipidemia, Former smoker: 40 pack-years, quit 2 years ago

Current Medications

Tiotropium inhaler once daily

Albuterol MDI as needed

Lisinopril 10 mg daily

Atorvastatin 20 mg nightly

Vitals: 100.6 *F, 112 bpm, 28/min labored, 152/88 mmHg, 86% on room air. Client is ALOx3. Client has diminished breath sounds with expiratory wheezing and accessory muscle use, prolonged expiratory phase. Productive cough with thick, green sputum. Heart rate 112bpm, regular rhythm. Bowel sounds present in all four quadrants. Voiding concentrated yellow urine with no complaints of pain. Skin clean, dry, and intact. Clubbing of fingernails present. 20G peripheral IV inserted into the right antecubital.

ABGsResultsReference
pH7.317.35-7.45
PaCO25835-45 mmHg
HCO33022-26 mEq/L
PaO25575-100 mmHg
CBCResultsReference
White blood cells14.54000-11000/mcL
Red blood cells5.8 million/mcL4.7-6.1 million/mcL
Hemoglobin13.1 g/dL13.2-16.6 g/dL
Hematocrit38%38.3-48.6%
Platelets152150-400 K/uL

Which provider orders should the nurse anticipate for a client with an acute COPD exacerbation? (Select all that apply)


Answer Options

Hint : ♦️ Bronchodilate, reduce inflammation, treat infection

Correct Answers:

  • Start empiric antibiotic therapy
  • Administer a short-acting bronchodilator
  • Administer corticosteroids intravenously


Rationale :

✅ Start empiric antibiotic therapy

♦️ The client has increased dyspnea, increased sputum production, and purulent green sputum, which strongly support an infectious trigger for the COPD exacerbation.

♦️ Purulent sputum is an important finding that increases the likelihood that bacterial infection is contributing to the acute deterioration.

♦️ The client's WBC count of 14.5 is elevated and supports an active inflammatory or infectious process.

♦️ The temperature of 100.6°F provides additional evidence suggesting infection.

♦️ Antibiotics are commonly indicated during an acute COPD exacerbation when there is increased sputum purulence, particularly when accompanied by worsening dyspnea or sputum volume.

♦️ Treating the infectious trigger can reduce continued airway inflammation and mucus production.

♦️ The chest x-ray showing no focal consolidation makes pneumonia less apparent, but it does not rule out a bacterial COPD exacerbation.

♦️ Therefore, the nurse should anticipate empiric antibiotic therapy based on this client's clinical presentation.

✅ Administer a short-acting bronchodilator

♦️ A short-acting bronchodilator, such as albuterol, is a first-line treatment for acute bronchospasm during a COPD exacerbation.

♦️ The client's expiratory wheezing indicates significant narrowing of the lower airways.

♦️ Bronchodilation helps decrease airway resistance and improves expiratory airflow.

♦️ The client's prolonged expiratory phase reflects airflow obstruction that can improve with short-acting bronchodilator therapy.

♦️ The presence of accessory muscle use and labored breathing demonstrates increased work of breathing that requires rapid treatment.

♦️ Short-acting beta₂ agonists begin working quickly and are appropriate for acute symptom relief.

♦️ Bronchodilator therapy can improve ventilation and help reduce CO₂ retention by improving airflow.

♦️ Therefore, administration of a short-acting bronchodilator should be anticipated promptly.

✅ Administer corticosteroids intravenously

♦️ Systemic corticosteroids decrease airway inflammation during moderate to severe acute COPD exacerbations.

♦️ Reduced inflammation can improve airflow and respiratory symptoms and shorten recovery time.

♦️ This client has substantial respiratory distress, including SpO₂ 86%, RR 28/min, accessory muscle use, and wheezing.

♦️ The ABG showing pH 7.31 and PaCO₂ 58 mmHg indicates significant ventilatory impairment.

♦️ Systemic corticosteroids can decrease airway edema and improve the effectiveness of bronchodilator therapy.

♦️ Intravenous administration may be used in a significantly symptomatic hospitalized client, although systemic corticosteroids can also be given orally when appropriate.

♦️ Because the client already has hyperglycemia risk related to stress and steroid therapy, glucose levels should be monitored carefully during treatment.

♦️ Therefore, systemic corticosteroid therapy is an expected component of treatment for this acute COPD exacerbation.


Distractor Analysis :

Rationale for Incorrect Options to be Available Soon

5 :

Chronic obstructive pulmonary disease (COPD) - diagnosed 10 years ago, Hypertension Hyperlipidemia, Former smoker: 40 pack-years, quit 2 years ago

Current Medications

Tiotropium inhaler once daily

Albuterol MDI as needed

Lisinopril 10 mg daily

Atorvastatin 20 mg nightly

Vitals: 100.6 *F, 112 bpm, 28/min labored, 152/88 mmHg, 86% on room air. Client is ALOx3. Client has diminished breath sounds with expiratory wheezing and accessory muscle use, prolonged expiratory phase. Productive cough with thick, green sputum. Heart rate 112bpm, regular rhythm. Bowel sounds present in all four quadrants. Voiding concentrated yellow urine with no complaints of pain. Skin clean, dry, and intact. Clubbing of fingernails present. 20G peripheral IV inserted into the right antecubital.

ABGsResultsReference
pH7.317.35-7.45
PaCO25835-45 mmHg
HCO33022-26 mEq/L
PaO25575-100 mmHg
CBCResultsReference
White blood cells14.54000-11000/mcL
Red blood cells5.8 million/mcL4.7-6.1 million/mcL
Hemoglobin13.1 g/dL13.2-16.6 g/dL
Hematocrit38%38.3-48.6%
Platelets152150-400 K/uL

 


Which nursing intervention is most appropriate during oxygen administration?


Answer Options

Hint : ✏️ COPD oxygen target is 88–92%

Correct Answers:

  • Titrate oxygen to maintain SpO2 between 88-92%


Rationale :

✏️ During an acute COPD exacerbation, supplemental oxygen should be carefully titrated, particularly in a client with evidence of CO₂ retention.

✏️ The client's SpO₂ of 86% and PaO₂ of 55 mmHg indicate significant hypoxemia, making supplemental oxygen necessary.

✏️ A target SpO₂ of 88–92% is commonly used for clients with COPD who are at risk for hypercapnic respiratory failure.

✏️ The client's PaCO₂ of 58 mmHg confirms significant carbon dioxide retention and supports the need for controlled oxygen administration.

✏️ Excessive oxygen can worsen hypercapnia in susceptible COPD clients through mechanisms including worsening ventilation-perfusion mismatch.

✏️ Oxygen should not be withheld because of concern for CO₂ retention; instead, it should be administered and carefully titrated to the prescribed target.

✏️ The nurse should continuously monitor SpO₂, respiratory effort, mental status, and respiratory rate, with repeat ABGs as clinically indicated.

✏️ Therefore, maintaining the client's SpO₂ between 88–92% provides needed oxygenation while reducing the risk associated with excessive oxygen administration.


Distractor Analysis :

Rationale for Incorrect Options to be Available Soon

6 :

Chronic obstructive pulmonary disease (COPD) - diagnosed 10 years ago, Hypertension Hyperlipidemia, Former smoker: 40 pack-years, quit 2 years ago

Current Medications

Tiotropium inhaler once daily

Albuterol MDI as needed

Lisinopril 10 mg daily

Atorvastatin 20 mg nightly

Vitals: 100.6 *F, 112 bpm, 28/min labored, 152/88 mmHg, 86% on room air. Client is ALOx3. Client has diminished breath sounds with expiratory wheezing and accessory muscle use, prolonged expiratory phase. Productive cough with thick, green sputum. Heart rate 112bpm, regular rhythm. Bowel sounds present in all four quadrants. Voiding concentrated yellow urine with no complaints of pain. Skin clean, dry, and intact. Clubbing of fingernails present. 20G peripheral IV inserted into the right antecubital.

ABGsResultsReference
pH7.317.35-7.45
PaCO25835-45 mmHg
HCO33022-26 mEq/L
PaO25575-100 mmHg
CBCResultsReference
White blood cells14.54000-11000/mcL
Red blood cells5.8 million/mcL4.7-6.1 million/mcL
Hemoglobin13.1 g/dL13.2-16.6 g/dL
Hematocrit38%38.3-48.6%
Platelets152150-400 K/uL

After 24 hours of treatment, which findings would indicate the client is improving? (Select all that apply)


Answer Options

Hint : ✨ Look for improving trends toward baseline

Correct Answers:

  • Temperature decreases from 100.6 °F to 99.5 °F
  • Arterial Blood Gas: pH 7.35, PaCO2 - 55, HCO, - 31
  • SpO2 increased from 86% to 90% on 2L nasal cannula


Rationale :

✅ Temperature decreases from 100.6°F to 99.5°F

✨ A decrease in temperature from 100.6°F to 99.5°F indicates improvement in the client's febrile response.

✨ The client's initial green sputum, elevated WBC count, and fever suggested an infectious or inflammatory trigger for the COPD exacerbation.

✨ A declining temperature after treatment suggests that the underlying infectious or inflammatory process may be responding to therapy.

✨ Improvement in infection can decrease airway inflammation and mucus production, helping improve respiratory function.

✨ A temperature of 99.5°F is much closer to the normal range than the client's initial temperature.

✨ This improvement should be considered together with respiratory findings, oxygenation, and laboratory values to determine the client's overall clinical response.

✨ Continued monitoring remains necessary because one improved temperature reading alone does not confirm complete resolution of the infection.

✨ Therefore, the decreasing temperature is a finding that indicates the client's condition is improving.

✅ Arterial blood gas: pH 7.35, PaCO₂ 55 mmHg, HCO₃⁻ 31 mEq/L

✨ The client's initial pH was 7.31, indicating acidemia from respiratory acidosis.

✨ An increase in pH to 7.35 demonstrates movement back into the normal range of 7.35–7.45.

✨ The PaCO₂ decreases from 58 to 55 mmHg, demonstrating some improvement in carbon dioxide elimination.

✨ The PaCO₂ remains elevated, which is not unexpected in a client with longstanding COPD and chronic CO₂ retention.

✨ The HCO₃⁻ of 31 mEq/L reflects renal bicarbonate retention associated with compensation for chronic respiratory acidosis.

✨ Normalization of the pH suggests that the client's overall acid-base status has improved compared with the initial ABG.

✨ The trend toward a lower PaCO₂ also supports improving alveolar ventilation following treatment.

✨ Therefore, these ABG values represent improvement from the client's initial pH 7.31 and PaCO₂ 58 mmHg.

✅ SpO₂ increased from 86% to 90% on 2 L nasal cannula

✨ The client's initial SpO₂ of 86% on room air demonstrated significant hypoxemia.

✨ An increase to 90% on 2 L/min nasal cannula demonstrates improved oxygenation following treatment.

✨ For many clients experiencing COPD exacerbations with risk for hypercapnia, oxygen is titrated toward approximately 88–92%.

✨ An SpO₂ of 90% falls within this commonly targeted range.

✨ Controlled oxygen administration improves tissue oxygenation while limiting unnecessary exposure to excessively high oxygen concentrations.

✨ Improvement in saturation suggests that interventions such as oxygen, bronchodilators, and corticosteroids are helping improve pulmonary function.

✨ The nurse should continue monitoring SpO₂, respiratory effort, mental status, and ABGs to ensure improvement continues.

✨ Therefore, increasing from 86% to 90% represents a clinically meaningful improvement in the client's oxygenation.


Distractor Analysis :

Rationale for Incorrect Options to be Available Soon

7 :

Hint : ✳️Acute asthma needs rapid bronchodilation

Correct Answers:

  • Albuterol


Rationale :

✳️ Albuterol is a short-acting beta₂-adrenergic agonist (SABA) used as a rescue medication during an acute asthma attack.

✳️ It produces rapid bronchodilation by relaxing bronchial smooth muscle and opening narrowed airways.

✳️ During an acute asthma attack, the immediate priority is to reverse bronchospasm and improve airflow.

✳️ Albuterol has a rapid onset of action, making it appropriate for immediate relief of wheezing, chest tightness, and shortness of breath.

✳️ The medication is commonly administered by metered-dose inhaler or nebulizer during an acute exacerbation.

✳️ The nurse should monitor for improvement in breath sounds, respiratory effort, oxygen saturation, and peak expiratory flow following administration.

✳️ Expected adverse effects can include tachycardia, palpitations, nervousness, and tremors because of beta-adrenergic stimulation.

✳️ Therefore, albuterol is administered first because it rapidly reverses the acute bronchoconstriction threatening the client's ventilation.


Distractor Analysis :

Rationale for Incorrect Options to be Available Soon

8 :

Hint : 💎 Previous intubation predicts severe asthma

Correct Answers:

  • Previous intubation due to status asthmaticus episode


Rationale :

💎 A history of previous intubation for status asthmaticus is a major risk factor for future severe or potentially fatal asthma exacerbations.

💎 Previous intubation indicates that the client's asthma has previously progressed to life-threatening respiratory failure.

💎 Status asthmaticus is a severe, persistent asthma exacerbation that does not respond adequately to initial standard bronchodilator treatment.

💎 Clients with a history of severe exacerbations are at increased risk for experiencing another episode of severe airway obstruction.

💎 Intubation is generally required when worsening bronchospasm results in respiratory exhaustion, severe hypoxemia, hypercapnia, or altered mental status.

💎 The nurse should therefore consider a history of previous intubation an important warning sign when assessing the client's risk for life-threatening asthma.

💎 These clients require careful monitoring and early aggressive treatment when manifestations of an acute asthma exacerbation develop.

💎 Therefore, previous intubation due to status asthmaticus is the strongest risk factor among the choices for another severe episode.


Distractor Analysis :

Rationale for Incorrect Options to be Available Soon

9 :

Hint : ⚽ Paralysis suppresses spontaneous respiratory effort

Correct Answers:

  • Suppress respiratory effort


Rationale :

⚽ Pancuronium is a nondepolarizing neuromuscular blocking agent that produces skeletal muscle paralysis.

⚽ In a mechanically ventilated client with ARDS, pancuronium can suppress spontaneous respiratory muscle activity and improve synchrony with the ventilator.

⚽ Severe ARDS may require controlled mechanical ventilation to optimize oxygenation and ventilation while limiting ventilator-induced lung injury.

⚽ By paralyzing the respiratory muscles, pancuronium prevents the client from fighting or breathing asynchronously with the ventilator.

⚽ Neuromuscular blockade can therefore suppress respiratory effort and allow the ventilator to provide controlled breaths more effectively.

⚽ Pancuronium causes paralysis only and does not provide analgesia, amnesia, or sedation.

⚽ Because the client can potentially remain conscious while paralyzed, adequate sedation and analgesia must be provided separately when neuromuscular blockade is used.

⚽ Therefore, pancuronium is administered in this situation to suppress respiratory effort and facilitate mechanical ventilation.


Distractor Analysis :

Rationale for Incorrect Options to be Available Soon

10 :

Hint : 🎯 Sepsis commonly triggers ARDS

Correct Answers:

  • Sepsis


Rationale :

🎯 Sepsis is the most common cause of acute respiratory distress syndrome (ARDS).

🎯 Sepsis triggers a widespread systemic inflammatory response that can injure the pulmonary capillary membrane.

🎯 This inflammatory injury causes increased capillary permeability, allowing fluid to leak into the alveoli.

🎯 The resulting noncardiogenic pulmonary edema interferes with normal oxygen exchange.

🎯 Clients with ARDS often develop severe hypoxemia that is difficult to correct with oxygen therapy alone.

🎯 Inflammation also reduces lung compliance, making the lungs stiff and increasing the work required for ventilation.

🎯 Sepsis-related ARDS can progress rapidly and often requires mechanical ventilation with lung-protective strategies.

🎯 Therefore, among the listed causes, sepsis is the most common precipitating condition for ARDS.


Distractor Analysis :

Rationale for Incorrect Options to be Available Soon

Total Questions: 10

Choose Different Exam !