End
1 :

Chronic obstructive pulmonary disease (COPD) - diagnosed 10 years ago, Hypertension Hyperlipidemia, Former smoker: 40 pack-years, quit 2 years ago

Current Medications

Tiotropium inhaler once daily

Albuterol MDI as needed

Lisinopril 10 mg daily

Atorvastatin 20 mg nightly

Vitals: 100.6 *F, 112 bpm, 28/min labored, 152/88 mmHg, 86% on room air. Client is ALOx3. Client has diminished breath sounds with expiratory wheezing and accessory muscle use, prolonged expiratory phase. Productive cough with thick, green sputum. Heart rate 112bpm, regular rhythm. Bowel sounds present in all four quadrants. Voiding concentrated yellow urine with no complaints of pain. Skin clean, dry, and intact. Clubbing of fingernails present. 20G peripheral IV inserted into the right antecubital.

ABGsResultsReference
pH7.317.35-7.45
PaCO25835-45 mmHg
HCO33022-26 mEq/L
PaO25575-100 mmHg
CBCResultsReference
White blood cells14.54000-11000/mcL
Red blood cells5.8 million/mcL4.7-6.1 million/mcL
Hemoglobin13.1 g/dL13.2-16.6 g/dL
Hematocrit38%38.3-48.6%
Platelets152150-400 K/uL

Which clinical findings are most consistent with an acute COPD exacerbation? (Select all that apply)


Answer Options

Hint : 🌹 COPD worsens expiration and oxygenation

Correct Answers:

  • Prolonged expiratory phase with wheezing
  • Productive cough with yellow-green sputum
  • Sp02 86% on room air


Rationale :

✅ Prolonged expiratory phase with wheezing

🌹 COPD exacerbations cause worsening airflow obstruction, making it particularly difficult for the client to exhale effectively.

🌹 A prolonged expiratory phase is characteristic of obstructive pulmonary disease because narrowed airways slow the movement of air out of the lungs.

🌹 Expiratory wheezing results from air moving through narrowed or obstructed lower airways.

🌹 Increased bronchospasm and airway inflammation during an exacerbation can make wheezing more pronounced.

🌹 The client's diminished breath sounds further indicate decreased air movement associated with significant airflow obstruction.

🌹 The ABG showing PaCO₂ 58 mmHg supports impaired ventilation and carbon dioxide retention.

🌹 The client's labored respirations and accessory muscle use further indicate increased work of breathing during the acute episode.

🌹 Therefore, a prolonged expiratory phase with wheezing is strongly consistent with an acute COPD exacerbation.

✅ Productive cough with yellow-green sputum

🌹 An acute COPD exacerbation commonly presents with an increase in cough and sputum production.

🌹 A change in sputum characteristics, particularly development of purulent yellow-green sputum, can suggest an infectious trigger.

🌹 Respiratory infections are common precipitating factors for acute COPD exacerbations.

🌹 The client's low-grade fever of 100.6°F further supports the possibility of an acute infectious process.

🌹 The elevated WBC count of 14.5 also indicates an inflammatory or infectious response.

🌹 Increased airway secretions can worsen airflow obstruction and contribute to difficulty breathing.

🌹 The absence of focal consolidation on the chest x-ray makes focal pneumonia less evident, while the overall presentation remains consistent with COPD exacerbation.

🌹 Therefore, the client's productive cough with yellow-green sputum is an important finding associated with the acute exacerbation.

✅ SpO₂ 86% on room air

🌹 An SpO₂ of 86% on room air represents significant hypoxemia and is consistent with worsening gas exchange during an acute COPD exacerbation.

🌹 The client's ABG confirms hypoxemia with a PaO₂ of 55 mmHg.

🌹 Increased airway obstruction and ventilation-perfusion mismatch can significantly reduce oxygenation during an exacerbation.

🌹 The client's tachypnea of 28/min represents a compensatory response to respiratory compromise.

🌹 Accessory muscle use further demonstrates increased respiratory effort and difficulty maintaining adequate ventilation.

🌹 The ABG values of pH 7.31, PaCO₂ 58 mmHg, and HCO₃⁻ 30 mEq/L indicate respiratory acidosis with metabolic compensation, compatible with chronic CO₂ retention with acute worsening.

🌹 Significant hypoxemia requires prompt assessment and appropriately controlled supplemental oxygen therapy in a client with COPD.

🌹 Therefore, SpO₂ 86% on room air is a major clinical finding consistent with an acute COPD exacerbation.


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